The importance of Flaxseed Oil taken on a daily basis.
Would it not be better to take the actual Flax seed, since the Flaxseed oil can go rancid?
Flaxseed / Linseed Oil for Liver and
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QUESTION:
The importance of Flaxseed Oil taken on a daily basis.
Would it not be better to take the actual Flax seed, since the Flaxseed oil can go rancid?
Answer
Yes , it is very good to use flaxseeds / linseeds.
Use it every day.
It is the best freshly grounded.
Some people first soak it, and then ground.
It is not very good to use it whole, unless you are ready to chew every mouthful over 100
times.
People doing Bowel cleanse have reported finding a lot of whole flax seeds, sesame seeds,
popcorn (unpoped) and other seeds.
It stuck in the bowel mucus, if it is not chewed well.
Some people have reported finding popcorn many years old, in the thing that came out while
bowel cleansing. (they remember not eating popcorn for many years.)
Flaxseed oil have one great advantage:
To get the same amount of Omega 3 fatty acid that you get from the 1 tbsp. of flaxseed
oil, you would have to eat at least 5 tbsp. of flaxseed.
And that is a big amount. It is not unhealthy to eat 5 - 10 tbsp. of flaxseed a day, but
it is unusual. I do not know anyone who do that.
And, what about people who need 5 tbsp. of flaxseed oil ?
Flaxseed oil is not very important for people who eat a lot of raw food, vegetable,
sprouts, don't eat sugar and other foods that kill.
Those people get enough Omega 3 , in good balance with Omega 6.
Flaxseed oil is important for people who don't live that kind of life.
LNA = Alpha Linolenic Acid (fatty acid), essential nutrients
LA = Linoleic Acid (fatty acid), essential nutrients
Modern diet have one great imbalance:
Ratio: Omega3 : Omega 6
LNA : LA
---------------------------------------
Our total needs: 1 : 2
---------------------------------------
Modern diet 1 : 10 -50
---------------------------------------
Flaxseed oil 4 : 1
---------------------------------------
Udo's Choice 2 : 1
---------------------------------------
hemp oil 1 : 3
---------------------------------------
soybean 1 : 7
---------------------------------------
safflower 0 : 75
sunflower 0 : 65
corn 0 : 59
Olive oil 0 : 8
canola
(Canadian rape) 1 : 4
Look for other oils here:
http://curezone.com/foods/fatspercent.asp
LA (omega 6 Linoleic essential fatty acid(EFA))
Polyunsaturated
Polyunsaturated means many times Unsaturated
(Two double bonds, four H are "missing")
Chemical formula:18:2w6
LA ( 18:2W6 ) C18 H32 O OH
Empirical formula Linoleic ACID:
SEE DOUBLE BONDS(=)
melting point 23 degrees F (-5C)
Linoleic Acid (LA) and is poly-unsaturated. LA is an 18 carbon chain
fatty acid with two double bonds in the middle and is missing four hydrogen
atoms, all on one side. The first double bond occurs after the 6th carbon
atom(W6), the second occur after the 9th carbon atom.
Because of this, LA is sometimes referred to as the W6(omega6) EFA.
Because of the four missing hydrogen atoms, LA is more bent than Oleic
Acid, giving it a melting temperature of 23 degrees F (-5C). LA is fairly
unstable, reacting with light and oxygen.
It is bent. It bends because of the opposing charges of the hydrogen atoms.
If you have ever played with magnets, you have recognized that the North
and South poles of two magnets pull towards each other. But when you turn
one of the magnets over and try to put the two North poles or the two South
poles together, they push apart. The same thing happens here. The hydrogen
atoms carry a negative charge and repel each other.
It is liquid in room temperature
LNA (omega 3 Alpha Linolenic essential fatty acid(EFA))
Poly-unsaturated (Super-unsaturated)
Super-unsaturated means very many times Unsaturated
(Three double bonds, six H are "missing")
Chemical formula:18:3w3
LNA ( 18:3W3 )
Empirical formula Alpha Linolenic ACID:
SEE 3 DOUBLE BONDS(=)
melting point 10 degrees F (-12 C)
Alpha Linolenic Acid (LNA). 18 carbon fatty acid with three double bonds
after 3rd, 6th, and 9th carbon positions. LNA is missing 6 hydrogen atoms,
again all from one side. Because of the first double bond occurring at the
3rd carbon atom, LNA is referred to as the W3 EFA.
It is also referred to as a super unsaturated fatty acid (SUFA), even
though it is also poly-unsaturated. Because LNA is bent more than the
others, it has the lowest melting point of the three, 10 degrees F (-12 C).
LNA is 5 times more unstable than LA and quickly goes rancid if exposed to
light or oxygen. It is so unstable, in fact, that when it is pressed from
the seeds that possess it, the pressing must be done in the total absence
of light and oxygen. It must be handled in this way right through to the
packaging stage, then quickly refrigerated or frozen.
Essential: LA and LNA are essential in our diets because our bodies cannot
make them, yet use them to make many other fatty acids our bodies must
have. Therefore, without LA and LNA, our bodies miss out on a bunch of
fatty acids we must have to be healthy.
References: "Fats That Heal, Fats That Kill" by
Dr.Udo Erasmus
As you can see, oils that can balance great deficiency of Omega3 in
modern diet are oils that contain a lot of Omega 3:
Flaxseed oil
Hemp oil
Udo's Choice (contain at least 50% flaxseed oil)
canola
soybean
Flaxseed is the richest source of Omega 3 LNA.
Flaxseed oil increases immunity, it makes nails and hair grow better,
and it makes skin softer:
Alpha Linolenic Acid (LNA)
Deficiency Symptoms:
growth retardation;
weakness;
impairment of vision and learning ability;
motor incoordination;
tingling sensations in arms and legs;
behavioral changes;
These symptoms can be reversed by adding LNA back to the diet from which
it was missing. Other symptoms that can result from LNA (or w3)
deficiency include :
high triglycerides;
high blood pressure;
sticky platelets;
tissue inflammation;
edema;
dry skin;
mental deterioration;
low metabolic rate;
some kinds of immune dysfunction;
These are not considered 'classic' symptoms of w3 deficiency, but often
respond remarkably well to w3 supplementation.
LNA is five times more unstable than LA and modern people consume 10
times less LNA then LA.
LNA is found in flax, hemp seed, canola(rape seed), soybean, walnut and
dark-green leaves. Flax seed is the richest source, containing over 50%
of its fatty acids as LNA. Chia and kukui (candlenut) oils contains
about 30% LNA. Hemp seed oil contains about 20% LNA. Pumpkin seed oil
contains between 1% and 15% LNA. Canola oil contains up to 10% LNA and
Walnut oil contains between 3% and 11% LNA.
How to keep oil from getting rancid ?
When buying, buy only fresh: It must stay frozen or in the fridge of The
Health food shop. If kept outside fridge, it rapidly turn rancid.
Educate people in the Health food shop, if they are not keeping it
frozen or in the fridge.
Frozen oil can keep fresh for years. It freezes on ~-7degree Celsius.
When using at home, close the bottle after use and put it back in the
freezer. Like that it can keep 2-3 weeks , and still taste good.
If rancid, use it for painting and preserving wood or threw it, don't
eat it.
Fresh flaxseed oil can cure or help curing cancer.
Rancid flaxseed oil contain carcinogens. (You can get cancer if eating
rancid flaxseed oil)
Buy always small bottle, rather then big. You will use up small bottle
faster then big.
Once opened, air comes inside the bottle, and oxidation starts.
If you don't use it up within 2 -3 weeks, it is rancid.
Read here more about flaxseed oil:
~--------------------------------------
Date: Sun, 03 Jan 1999 14:37:37 -0500
From: PJ Macon <pjmacon@ix.netcom.com>
To: "cancercure@egroups.com" <cancercure@egroups.com>
Subject: [cancercure] Colon Cancer and Flax (Lignans)
Here is some current research to back up the flax seed/oil
recommendations. Using flax seed in cottage cheese is not an options
for me since the casein protein in dairy products causes my rheumatoid
arthritis to flare.
I use flax seed in Bob's Red Mill's eight grain cereal. I cook the
cereal per directions and as it cools I add 2 tbsp. of ground flax seed
and 1 tbsp. of Barleans oil. Along with cinnamon, honey and some organic
raisins it makes a tasty breakfast that my husband will eat without
complaining!
Below are some more Medline studies regarding the use of flax and colon
cancer:
Regards,
Pamela
Anticancer Res 1998 May-Jun;18(3A):1405-8&;
Mammalian Lignans inhibit the growth of
estrogen-independent human colon tumor cells.
Sung MK, Lautens M, Thompson LU
Department of Nutritional Sciences, University of Toronto, Ont.,
Canada.&nabs;
PURPOSE: The mammalian Lignans enterolactone (EL) and enterodiol (ED) derived from
precursors in foods, particularly flaxseed, have been shown to reduce the mammary tumor
growth due to their antiestrogenic properties. Our objective was to investigate whether
lignans have growth inhibitory effects against colon tumor cells and whether these effects
are mediated through their antiestrogenic
activity.
METHODS: Four human colon tumor cell lines (LS174T, Caco-2, HCT-15, T-84) were incubated
with various levels of EL, ED or 17 beta-estradiol for 8 to 10 days. Cell
proliferation was measured by the BrdU method.
RESULTS: At 100 microM concentration, both lignans significantly reduced the proliferation
of
all cell lines. EL was more than twice as effective as ED at this concentration. The
growth was not
affected by the presence of 17 beta-estradiol implying that these cells are not
estrogen-sensitive.
CONCLUSION: Lignans are growth inhibitors of colon tumor cells and they may act through
mechanism(s) other than antiestrogenic activity.
Carcinogenesis 1996 Jun;17(6):1343-8&;
The influence of flaxseed and lignans on colon carcinogenesis and beta-glucuronidase
activity.
Jenab M, Thompson LU
Department of Nutritional Sciences, Faculty of Medicine, University of Toronto, Ontario,
Canada.&;
Flaxseed, the richest source of mammalian lignan precursors, such as secoisolariciresinol
diglycoside (SD), has been shown over the short term to decrease some early markers of
colon cancer risk. This study determined whether over the long term flaxseed still exerts
a colon cancer protective effect,
whether its effect may, in part, be due to its high content of SD and whether any change
in beta-glucuronidase activity plays a role in the protective effect.
Six groups of male Sprague-Dawley rats were fed for 100 days either a basal high fat (20%) diet (BD), BD supplemented with 2.5 or 5% flaxseed or 2.5 or 5% defatted flaxseed (equivalent to the respective flaxseed diets) or BD with a daily gavage of 1.5 mg SD.
All rats were injected with a single dose of
azoxymethane (15 mg/kg body wt) 1 week prior
to commencing the dietary treatments. Urinary lignan excretion, which is an indicator of
mammalian lignan production, was significantly increased in the flaxseed and defatted
flaxseed groups. The total activity of cecal beta-glucuronidase was significantly
increased in a dose-dependent manner by the flaxseed and defatted flaxseed diet groups.
Compared with the control the number of aberrant
crypts per focus was significantly reduced in the distal colon of the treated rats.
Four microadenomas and two polyps were observed
in the control group, but not in the treated groups. The total activity of
beta-glucuronidase was positively correlated with total urinary lignan excretion and
negatively with the total number of aberrant crypts and the total number of aberrant crypt
foci in the distal colon. There were no significant differences between the flaxseed and
the corresponding
defatted flaxseed groups. It is concluded that flaxseed has a colon cancer protective
effect, that it is
due, in part, to SD and that the protective effect of flaxseed is associated with
increased beta-glucuronidase activity.
Pamela
http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9673380&form=6&db=m&Dopt=b
Anticancer Res 1998 May-Jun;18(3A):1621-7
Dietary effects of fatty acids on growth and
metastasis of KPL-1 human breast cancer cells in
vivo and in vitro.
Senzaki H, Iwamoto S, Ogura E, Kiyozuka Y, Arita S, Kurebayashi J,
Takada H,
Hioki K, Tsubura A
Department of Pathology II, Kansai Medical University, Osaka, Japan.
To evaluate the effects of dietary fats on breast cancer growth and metastasis, KPL-1
human breast carcinoma cells which have a propensity for axillary lymph node metastasis
when inoculated into the thoracic mammary fat pad of female nude mice were examined. The
mice were fed one of three semipurified diets containing 9.5% eicosapentaenoic acid plus
0.5% linoleic acid (EPA diet), 10% linoleic
acid (LA diet), or 9.5% palmitic acid plus 0.5% linoleic acid (PA diet), or commercial
laboratory chow containing 8.5% fat of which 4.1% was LA, 1.1% was PA, 0.06% was EPA, and
3.24% was other (Standard diet) starting 19 days before tumor cell inoculation and
continuing until the end of the experiment (43 days after tumor cell inoculation).
The tumor growth was faster and at a higher incidence in the mice fed the LA diet, and
much slower and at a lower incidence in the EPA diet group compared with the mice fed the
PA or Standard diet; the two separate experiment demonstrated identical results. The
differences in tumor weight between the LA and PA groups and between the PA and EPA groups
were significant (P < 0.05, respectively) at the
termination of the experiment; the differences were due to different tumor cell
proliferation rates. In
an in vitro MTT assay, fatty acids showed direct stimulatory or inhibitory effects on the
KPL-1 cells. Lymph node metastasis was seen in the LA and Standard diet groups, whereas it
was not seen in the PA or EPA groups. The body weights were significantly lighter in the
LA and EPA groups compared with the PA and Standard diet groups (P < 0.05,
respectively).
The results indicate that the EPA diet produced a reduction in tumor cell growth and
metastasis whereas the LA diet had an enhancing effect on these parameters; dietary fatty
acids may thus have a direct role in the growth and metastasis of human breast carcinoma
independent of their
systemic effects.
And more about flaxseed oil:
Read here: Dr Johanna Budwig diet
Today, fresh expeller pressed flaxseed oil is available in most
countries in the world, including USA and Europe.
GALLSTONES
CLEANSE FLUSH - AVOID GALLBLADDER SURGERY !
