PinX box dose is one dose every 6 weeks. I suggest one full dose = a PinX challenge test.
_muscle spasms/pherpherial neuropathy
Actions: ALB, PinX, PZQ with D3.
Ruled out: Nematode, Fluke, Flatworm, PinWorm.
_750mg PPZ or 1850mg - 2000mg nPPZ is safer choice
_Magnesium sulfate 1030mg/D
_Arginase Bladder (2)/D
If nitrogen pathway congestion, will see looser bowel, brief reduction in Ammonia load.
WBC Microbe Challenges:
If WBC white blood cell exhaustion like in ADD, There is one pathway that gets depleated.
Depletion of this pathway can be tested by performing a Kale challenge test:
Eat Kale (1/2) cup, be careful, can be challenging.
If Kale causes liver pain - Lyme flagallate
If Kale relievs pain - think SCN deficient, amoeba, bacterial other.
Illnesses that consume excessive white blood cells can be identified by challenges:
to see if nano microbe.
_Iodine 25,000 units, Terrys tri Iodine
or liquid Lugol
Solution provides 12.5 mg per 2 drops.
_600-800ucg Selenium chelate p for 2-4 weeks
Medical Challenge History:
Methimazole administration prevented the SCN-induced necrosis - See more at:
Enzyme, and or White blood cell exhaustions:
_B12 at 8000 IU/D for 2 weeks
_Coconut Butter caps
_Iodine 25000 tri Iodine
per day balances the thiocyanate pathways
_300 mg of phenytoin daily (med)
_or 1500mg Taurine for 3 days. (natural)
Both Liver congestion, and SCN congestion can be relieved by challenging Taurine.
Body Supplement test from ingredients above:
Supplement the following sequence:
kale increases thiocyanate, tiny amounts till log jam removed.
_Ramp B12, B50, Selenium, and Iodine, then gradually increase Kale.
Wild cherry (black cherry concentrate R255), Increase Choline, http://www.nutramedix.ec/ns/pinella
Bitter Apricot Kernels
B17 - apricot seeds (Bulk) - Each seed contains 17-20 mg of Amygdalin, the precursor/enabler of Thiocyanate. Dose teaspoon of seeds.
Body Process Loop Drag on SCN, Genes, DNA, other.
Sea Salt can be helpful over iodine salt. Iodine diff is common in many countries.
Iodine deficiency (ID) and related disorders are still major, yet unresolved health concerns. Recently, in a
systematic survey of schoolage children (SAC), we reported severe to moderate ID, in Ankara and three cities from Black Sea region of Turkey. The current study attempted to evaluate selenium (Se) status, thiocyanate (SCN−) overload, and their possible contribution to the goiter endemics and thyroid hormone profile observed in these cities. Thyroid ultrasonography was performed and serum Se, SCN−, thyroid hormones, sensitive TSH (sTSH) levels, and urinary iodine concentrations (UICs) were determined from 251 SAC (9–11 yr old).
Thyroid volumes (TVs) exceeding recommended upper normal limits and median UIC indicated goitre endemics and moderate to severe ID in the areas studied. Mean serum SCN− concentrations were found to be greater than the controls from the literature. The UIC/SCN− ratio was found to be lowest in Bayburt and Trabzon denoting that
SCN− overload may contribute to the goiter endemics. Serum Se concentrations represent a marginal deficiency in the four areas studied. No significant correlations between serum Se concentrations and the other parameters studied (i.e., TV, SCN−, thyroid hormones, sTSH, UIC) was detected.
The effect of protein deficiency or administration of antithyroid drugs or both on the thyroids of suckling and weanling rats was examined. Addition of cyanide or propylthiouracil to a 2% casein low iodine diet for weanling rats resulted in an increase in thyroid weight and a significant increase in plasma thyroid stimulating hormone. The effect of cyanide was eliminated by addition of iodine to the diet and was not apparent when the animals were fed a 20% casein diet. Addition of thiocyanate (SCN -) to the drinking water of iodine deficient rats resulted in the appearance of goiters in both the mothers and their 5- and 10-day old progeny. Studies with 35SCN - in these animals indicated that the concentration of plasma 35SCN - decreased when exogenous SCN - was provided; the fractional transfer of 35SCN - into their milk and to the pups decreased, as did the conversion of 35SCN - to 35SO 4=. In adult male rats addition of thiocyanate to the ration increased renal disposal of both labeled and stable SCN -. These findings are consistent with saturation of renal tubular reabsorption and metabolism of SCN -. These studies support the proposition that cyanogenic glycosides and thiocyanate may affect thyroid function in the neonatal iodine deficient rat. They do not support the hypothesis that protein deficiency is a major factor in the responses of the thyroid to iodine deficiency or goitrogens.
Not Ruled out:
Giardia - gas
Flagallate (malaria, VL, Mountain Diseases)
Source of Non-focal nerve interruptions.
Next stop: Neurology