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Re: TSH and AF?
 

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Published: 11 years ago
 
This is a reply to # 1,662,992

Re: TSH and AF?


Yes, I have high TSH and also hypoadrenia. Doc put me on 1/2 grain of Pathroid back in Feb. I have not been able to see any improvement, though. Then, started on Cortef for hypoadrenia a little over a month ago. That has it's ups and downs, but I have noticed that I have a lot more fatigue in the last couple of months. I read the article posted above on Dr. Lam's site about hypothyroid vs. adrenal fatigue. He says there that if you treat the hypothyroid with medication and don't see improvements, it could be because of the adrenal issue. He also says that if you treat the hypothyroid with medication that it can actually cause the adrenal situation to get worse (if you are not treating the adrenals) because the metabolic rate is speeding up, but the adrenals are trying to slow down.

So, all that to say, does anyone have opinions or thoughts on whether I should stop taking my Pathroid for awhile while I work on my adrenals only? I am wondering if the Pathroid is making me more exhausted and that maybe that is why I don't see much energy improvement with the Cortef. But after starting the Cortef, my hair is not falling out near as much and my body pain is much better.

Below is an excerpt from the article:

In such cases, laboratory test results of T4 and T3 may be normal and classic symptoms of hypothyroid are evident with persistent low body temperature and slow ankle reflex. Alternatively, laboratory test results of free T4 and free T3 may be low while the TSH level is normal or high. In both scenarios, thyroid replacement with T4 and T3 without first considering adrenal fortification is a common mistake and often leads to a worsening state of adrenal fatigue over time. The reason is simple. Thyroid replacements tend to increase metabolic function and energy output. Raising the basal metabolic rate is akin to putting all systems of the body into overdrive at a time when the body is trying to rest by down-regulation through the many mechanisms described above. The body's survival mechanism is designed to achieve a reduction of and not the increase in the levels of T4 and T3. What the body wants (to slow down) and what the medications are designed to do (to speed up) is diametrically opposed to one another.
 

 
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