>>>So for starters what she is describing IS NOT an anxiety attack as the doctors stated.<<<
So, even though...
---this person has had gallbladder attack/s in the past and certainly knows what one feels like (but did NOT think she was experiencing a gallbladder attack, and would likely have known if she was)
--the doctor in the ER diagnosed a panic/anxiety attack, and it was relieved by Ativan/Morphine (Ativan being a benzodiazepine which is commonly & effectively used for panic/anxiety)
--the oil was ingested the night before this event (likely around 10-12 hours before), and anyone that's experienced a gallbladder attack knows it happens very quickly after ingesting fats & oils.
--you've never met this person, seen their labs and know nothing more than you've read in a very short posting history...
Even with all this, you KNOW this was NOT an anxiety attack and claim it was a gallbladder attack caused by flushing.
You consider this type of "reasoning" to be valid (?), while questioning and ridiculing the same of others.
And for what it's worth, not everyone agrees with your lactate/anxiety theory:
JURGEN MARGRAF, DIPL-PSYCH, ANKE EHLERS, DIPL-PSYCH, AND
WALTON T. ROTH, MD
www.psychosomaticmedicine.org/content/48/1/23.full.pdf Response to sodium lactate infusions has been proposed as an experimental model and a biologic
marker for panic attacks. Several authors have claimed that patients suffering from panic attacks,
but not normal controls, "panic" in response to lactate. A careful review of methods and results
of 13 studies, however, reveals serious methodologic problems, lack of specificity and sensitivity,
and a failure to consider cognitive variables. When baseline differences are ruled out,
the responses of patients and controls may not differ. So far, response to lactate cannot be
interpreted as a model and marker for panic attacks and does not provide evidence for their
underlying biologic distinctness from other types of anxiety. Known biologic mechanisms do
not sufficiently explain the effects of lactate. Instead, an interaction of peripheral physiologic
changes, past experience, environmental cues, and their appraisal as threatening or dangerous
seems to be a more appropriate model.
::: sitting back in my chair, getting ready to 'click off' the inevitable 'recording' about what's wrong with me and my reasoning, what's wrong with what I know & believe, how many times you've proven I am wrong, and how that's the reason I'm 'attacking' you :::