The original DNA defect I found was with Tcn1 gene, which binds with femtomolar affinity and protects it from the acidic environment of the stomach. This is a strong red flag in my DNA with a rare frequency rate of 0%. Hence, there's likely an issue with B12 not surviving the acidity of my stomach. Maybe B12 shots would help with this issue. However, B12 shots might not work for the other MTHFR/COMT gene defect because my body has trouble converting regular B12 or absorbing it. That's where the coenzyme B Complex supplements come in handy. It's puzzling how these supplements get absorbed if my stomach acid destroys B12 (without the Tcn1 protection other people have). Maybe because it's a different form of B12? At this point, I don't yet know if my B12 level will ever reach a normal level, but I'd still need to keep taking the B supplements regardless.