Re: I cured my Candida forever
I was able to find this information about the low CD8 T-Suppressor/Cytotoxic cells.
The information below suggests that vitamin D deficiency contributes to the development of autoimmune diseases by depleting CD8+ T cells.
"CD8+ T cells in the blood are decreased and CD4/CD8 ratio is increased in patients with autoimmune diseases, including multiple sclerosis, Rheumatoid Arthritis
, SLE, SjŲgrenís syndrome, systemic sclerosis, dermatomyositis, primary biliary cirrhosis, primary sclerosing Cholangitis
, ulcerative colitis, Crohnís disease, psoriasis, vitiligo, bullous pemphigoid, alopecia areata, idiopathic dilated cardiomyopathy, type 1 diabetes mellitus, Gravesí disease, Hashimotoís thyroiditis, myasthenia gravis, IgA nephropathy, membranous nephropathy (or membranous glomerulonephritis), and pernicious anaemia. Although some studies have not found CD8+ T-cell deficiency in patients with autoimmune diseases, CD8+ T-cell deficiency would appear to be a general feature of human chronic autoimmune diseases.
Exposure to natural sunlight increases the proportion of CD8+ T cells and decreases the CD4/CD8 T cell ratio in blood .
Exactly how sunlight increases the number of CD8+ T cells is unclear, but the effect is probably mediated at least in part by vitamin D because (1) among cells of the immune system, activated CD8+ T cells express the highest concentrations of the vitamin D receptor]; (2) vitamin D increases the mitogen-induced proliferation of CD8+ T cells and decreases the CD4/CD8 ratio in bovine blood mononuclear cells in vitro; (3) vitamin D administration increases the CD8+ T cell count ; (4) vitamin D deficiency is associated with a decreased proportion of CD8+ T cells and increased CD4/CD8 ratio. Here it is proposed that deprivation of sunlight and vitamin D aggravates the genetically determined CD8+ T cell deficiency and thereby contributes to the high prevalence of autoimmune diseases and that the protective effect of vitamin D against autoimmune diseases is mediated at least in part by an increase in the number of CD8+ T cells available."